Faculty Profiles - MITSUI Takashi

MISC - MITSUI Takashi

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P3-52-4 不育症に対する妊娠早期の低用量アスピリン投与(ASA)の妊娠高血圧症候群の予防効果について(Group 117 妊娠高血圧症候群3,一般演題,公益社団法人日本産科婦人科学会第68回学術講演会)

平野友美加, 増山寿, 江口武志, 玉田祥子, 光井崇, 衛藤英理子, 延本悦子, 早田桂, 酒本あい, 小谷早葉子, 鎌田泰彦, 平松祐司

日本産科婦人科學會雜誌 68 ( 2 ) 985 - 985 2016

MW5-1 "Diabetes begets diabetes" : メカニズムの解明に向けて(ミニワークショップ5 妊娠の栄養代謝,一般演題,公益社団法人日本産科婦人科学会第68回学術講演会)

増山寿, 江口武志, 玉田祥子, 光井崇, 平野友美加, 衛藤英理子, 延本悦子, 早田桂, 瀬川友功, 平松祐司

日本産科婦人科學會雜誌 68 ( 2 ) 600 - 600 2016

P2-56-9 妊娠高血圧症候群の発症時期による新生児,乳幼児期の発育への影響(Group56 妊娠高血圧症候群1,一般演題,公益社団法人日本産科婦人科学会第68回学術講演会)

光井崇, 増山寿, 牧尉太, 江口武志, 玉田祥子, 平野友美加, 衛藤英理子, 延本悦子, 早田桂, 平松祐司

日本産科婦人科學會雜誌 68 ( 2 ) 787 - 787 2016

P2-46-6 当院で経験した心疾患合併妊娠の検討(Group46 合併症妊婦1,一般演題,公益社団法人日本産科婦人科学会第68回学術講演会)

松原侑子, 延本悦子, 鈴井泉, 光井崇, 平野友美加, 衛藤英理子, 早田桂, 瀬川友功, 増山寿, 平松祐司

日本産科婦人科學會雜誌 68 ( 2 ) 750 - 750 2016

P3-41-2 抗NMDA受容体抗体脳炎による心肺停止から回復後,自然妊娠し生児を得た一例(Group106 合併症妊娠8,一般演題,公益社団法人日本産科婦人科学会第68回学術講演会)

玉田祥子, 牧尉太, 江口武志, 光井崇, 平野友美加, 衛藤英理子, 延本悦子, 早田桂, 瀬川友功, 増山寿, 平松祐司

日本産科婦人科學會雜誌 68 ( 2 ) 943 - 943 2016

糖代謝異常妊婦の安静時代謝量と体組成

衛藤英理子, 岡本和浩, 柏原麻子, 牧尉太, 江口武志, 玉田祥子, 光井崇, 早田桂, 増山寿, 平松祐司

日本産科婦人科栄養・代謝研究会プログラム・抄録集 40th 2016

妊娠高血圧症候群を合併した母体の出生児の3歳までの身体発育の性差

光井崇, 増山寿, 江口武志, 玉田祥子, 衛藤英理子, 早田桂, 平松祐司

日本産科婦人科栄養・代謝研究会プログラム・抄録集 40th 2016

“Diabetes begets diabetes”-メカニズムの解明に向けて-

増山寿, 江口武志, 玉田祥子, 光井崇, 平野友美加, 衛藤英理子, 延本悦子, 早田桂, 瀬川友功, 平松祐司

日本産科婦人科学会雑誌 68 ( 2 ) 2016

TWO CASES OF CONGENITAL CYTOMEGALOVIRUS INFECTION

Shoko Tamada, Jota Maki, Takeshi Eguchi, Takashi Mitsui, Yumika Hirano, Eriko Eto, Etsuko Nobumoto, Kei Hayata, Hisashi Masuyama, Yuji Hiramatsu

PLACENTA 36 ( 10 ) A3 - A4 2015.10

A case of pregnancy-associated gastric cancer diagnosed due to the occurrence of acute pancreatitis

64 ( 2 ) 431 - 434 2015

Two cases of fetal skeletal dysplasia

64 ( 2 ) 159 - 164 2015

The study of fetal congenital heart disease in our hospital

64 ( 2 ) 229 - 233 2015

P2-58-1 18トリソミーに特徴的な小脳低形成検出を目的とした,小脳横径と小脳半球縦径の基準値作成と臨床応用(Group 99 胎児異常1,一般演題,公益社団法人日本産科婦人科学会第67回学術講演会)

早田桂, 渋川昇平, 光井崇, 平野友美加, 衛藤英理子, 延本悦子, 瀬川友功, 増山寿, 平松祐司

日本産科婦人科學會雜誌 67 ( 2 ) 805 - 805 2015

Different fetal and neonatal growth between early- and late-onset preeclampsia.

Mitsui T, Masuyama H, Eto E, Nobumoto E, Hayata K, Hiramatsu Y

Open Journal of Obstetrics and Gynecology 5 ( 9 ) 516 - 521 2015

The Effects of High-Fat Diet Exposure In Utero on the Obesogenic and Diabetogenic Traits Through Epigenetic Changes in Adiponectin and Leptin Gene Expression for Multiple Generations in Female Mice

Hisashi Masuyama, Takashi Mitsui, Etsuko Nobumoto, Yuji Hiramatsu

ENDOCRINOLOGY 156 ( 7 ) 2482 - 2491 2015

Language：English Publisher：ENDOCRINE SOC

Recent studies demonstrate that epigenetic changes under malnutrition in utero might play important roles in transgenerational links with metabolic diseases. We have previously shown that exposure to a high-fat diet (HFD) in utero may cause a metabolic syndrome-like phenomenon through epigenetic modifications of Adiponectin and Leptin genes. Because an association of obesity between mother and offspring endured in multiple generations, we examined whether HFD exposure in utero might affect the metabolic status of female offspring through multigenerational epigenetic changes of Adiponectin and Leptin genes and whether a normal diet in utero for multiple generations might abolish such epigenetic changes after exposure to a HFD in utero using ICR mice. We observed that the effect of maternal HFD on offspring over multiple generations in metabolic syndrome-like phenomenon such as weight and fat mass gain, glucose intolerance, hypertriglyceridemia, abnormal adiponectin and leptin levels, and hypertension, were accumulated with expression and epigenetic changes in Adiponectin and Leptin genes. A normal diet in utero in the subsequent generations after HFD exposure in utero diminished, and a normal diet in utero for 3 generations completely abolished, the effect of HFD in utero on weight and fat mass gain, insulin resistance, serum triglyceride, adiponectin, and leptin levels, with epigenetic changes of Adiponectin and Leptin genes. Exposure to a HFD in utero might affect glucose and lipid metabolism of female offspring through epigenetic modifications to Adiponectin and Leptin genes for multiple generations. Obesogenic and diabetogenic traits were abolished after a maternal normal diet for 3 generations.

DOI： 10.1210/en.2014-2020

Web of Science

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Creation of a cerebellar diameter reference standard and its clinical application to the detection of cerebellar hypoplasia unique to trisomy 18. International journal

Kei Hayata, Yuji Hiramatsu, Hisashi Masuyama, Eriko Etou, Etsuko Nobumoto, Takashi Mitsui

The journal of obstetrics and gynaecology research 41 ( 12 ) 1899 - 1904 2015

Language：English

AIM: We created a new reference standard focusing on the hemispheric anteroposterior cerebellar diameter (APCD) in addition to the transverse cerebellar diameter (TCD) and discussed whether or not the cerebellar measurement was useful for the detection of trisomy 18 (T18). MATERIAL AND METHODS: In 150 normal fetuses between 14 and 36 weeks of gestational age (GA), the TCD and APCD were prospectively measured. In 26 cases with T18, the value was compared with the control. RESULTS: At <22 weeks of gestation, the TCD reference standard was calculated as follows: TCD = (1.027 × GA) - 0.674 (R(2) = 0.97, P < 0.001). The reference standard of the APCD was calculated as follows: APCD = (0.682 × GA) - 3.925 (R(2) = 0.73, P < 0.001). In eight cases with T18, the TCD was below the 5th percentile value in 7/8 (88%) cases and the APCD was below the 5th percentile value in 8/8 (100%) cases. At >22 weeks of gestation, the reference standard of the TCD was calculated as follows: TCD = (1.603 × GA) - 13.216 (R(2) = 0.92, P < 0.001). The reference standard of the APCD was calculated as follows: APCD = (0.859 × GA) - 7.30 (R(2) = 0.84, P < 0.001). In 18 cases with T18, the TCD was below the 5th percentile value in 14/18 (78%) cases and the APCD was below the 5th percentile value in 18/18 (100%) cases. CONCLUSION: APCD reference standard, divided by the gestational age of more or less than 22 weeks, might be useful to diagnose T18.

DOI： 10.1111/jog.12814

PubMed

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GDMに対するSMBGの保険適応拡大に向けて.

平野友美加, 光井崇, 渋川昇平, 衛藤英理子, 延本悦子, 早田桂, 瀬川友功, 増山寿, 平松祐司

糖尿病と妊娠 15 ( 1 ) 76 - 76 2015

P1-33-8 妊娠中に顕在化した緩徐進行1型糖尿病の1例(Group33 合併症妊娠4,一般演題,公益社団法人日本産科婦人科学会第67回学術講演会)

清時毅典, 延本悦子, 光井崇, 衛藤英理子, 早田桂, 瀬川友功, 増山寿, 平松祐司

日本産科婦人科學會雜誌 67 ( 2 ) 622 - 622 2015

P3-35-6 妊娠17週にて子宮破裂を発症した既往帝王切開後妊娠の1例(Group137 子宮破裂 TOLAC,一般演題,公益社団法人日本産科婦人科学会第67回学術講演会)

光井崇, 西田傑, 中村圭一郎, 早田桂, 谷和祐, 河原義文, 衛藤英理子, 延本悦子, 瀬川友功, 増山寿, 平松祐司

日本産科婦人科學會雜誌 67 ( 2 ) 907 - 907 2015

先天性サイトメガロウイルス感染症の2例

玉田祥子, 牧尉太, 江口武志, 光井崇, 平野友美加, 衛藤英理子, 延本悦子, 早田桂, 増山寿, 平松祐司

日本絨毛性疾患研究会・日本胎盤学会学術集会プログラム・抄録集 33rd-23rd 2015

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